Publikationen vor 2012
Ottaviani E, Ventura N, Mandrioli M, Candela M, Franchini A, and Franceschi C. Gut microbiota as a candidate for lifespan extension: an ecological/evolutionary perspective targeted on living organisms as metaorganisms. Biogerontology. 2011 Dec;12(6): 599-609.
Guccini I, Serio D, Condò I, Rufini A, Tomassini B, Mangiola A, Maira G, Anile C, Fina D, Pallone F, Mongiardi MP, Levi A, Ventura N@, Testi R@, Malisan F@. Frataxin participates to the hypoxia-induced response in tumors. Cell Death Dis. 2011 Feb 24;2:e123.
Rufini A*, Fortuni S*, Arcuri G, Condo’ I, Serio D, Incani O, Malisan M, Ventura N, Testi R. Preventing the ubiquitin/proteasome-dependent degradation of frataxin, the protein defective in Friedreich’s Ataxia. Hum Mol Genet. 2011 Apr 1;20(7):1253-61
Butler JA, Ventura N, Johnson TE, Rea SL. The Long-lived mitochondrial (MIT) mutants of C. elegans utilize a novel metabolism. FASEB J. 2010 Dec;24(12):4977-88
Torgovnick A, Schiavi A, Testi R, Ventura N. A role for p53 in mitochondrial stress response control of longevity in C. elegans. Exp Gerontol. 2010 Aug;45(7-8):550-7
Ventura N@, Rea SL, Schiavi A, Torgovnick A, Testi R, Johnson TE. p53/cep-1 increases or decreases C. elegans lifespan depending on level of mitochondrial bioenergetic stress. Aging Cell. 2009 Aug;8(4):380-93
Rea SL*, Ventura N*, Johnson TE. Relationship Between Mitochondrial Inhibition, Development and Life Extension in C. elegans. PloS Biology. 2007 Oct 2;5(10):e259
Kell A, Ventura N, Johnson TE. Activation of Skn-1 by novel kinases in Caenorhabditis elegans. Free Radic Biol Med. 2007 Dec 1;43(11):1560-6.
Ventura N*@, Rea SL*. C. elegans mitochondrial mutants as an investigative tool to study human neurodegenerative diseases associated with mitochondrial dysfunction. Biotechnol J. 2007 May;2(5):584-95
Condò I, Ventura N, Malisan F, Rufini A, Tomassini B, Testi R. In vivo maturation of human frataxin. Hum Mol Genet 2007 Jul 1;16(13):1534-40
Condò I*, Ventura N*, Malisan F, Tomassini B, Testi R. A pool of extramitochondrial frataxin that protect from apopotsis. J Biol Chem. 2006 Jun16;281(24):16750-6
Ventura N, Rea SL, Henderson ST, Condo I, Testi R, Johnson TE. C. elegans as a model for Friedreich Ataxia. FASEB J 2006 May; 20(7):1029-30.
Ventura N@, Rea SL, Testi R. Long-lived C. elegans mitochondrial mutants as a model for human mitochondrial-associated diseases. Exp Gerontol 2006 Oct;41(10):974-91
Ventura N, Rea S, Henderson ST, Condo I, Johnson TE, Testi R. Reduced Expression of Frataxin Extends the Life Span of Caenorhabditis elegans. Aging Cell 2005 Apr;4(2):109-12.
Barilà D, Rufini A, Condò I, Ventura N, Dorey K, Superti-Furga G and Testi R. Caspase-dependent cleavage of c-Abl contributes to apoptosis. Mol Cell Biol 2003 Apr;23(8):2790-9.
Cicconi S, Ventura N, Pastore D, Bonini P, Pierucci D, Merlo D, Di Nardo P, Lauro R, Borboni P. Marlier NJL. Characterization of apoptosis signal transduction pathways in HL-5 cardiomyocytes exposed to ischemia/reperfusion oxidative stress model. J Cell Physiol 2003 Apr;195(1):27-37
Malisan F, Franchi L, Tomassini B, Ventura N, Condo' I, Rippo MR, Rufini A, Liberati L, Nachtigall C, Kniep B, Testi R. Acetylation suppresses the pro-apoptotic activity of GD3 ganglioside. J Exp Med 2002 196(12):1535-41
*equal contribution; @corresponding author